1. Signs and symptoms and management of musculoskeletal sprains/strains/dislocations KaraIrvin Signs and symptoms and management of musculoskeletal sprains/strains/dislocations Sprains: stretching or tearing of ligaments that occurs when a joint is forcedbeyond its normal anatomical range First degree- stretching of ligamentous fibers Second degree- tear of part of the ligament with pain and swellingThird degree- complete ligamentous separation Sprain- sudden injury or fall that caused acute pain and swelling that gotworse over a few hours, redness and bruising, active and passive ROM decreased. Radiography to rule out fx. Strain: muscle injury caused by excessive tensile stress placed on a musclethat results in stiffness and decreased function -effects muscle or tendon that connects a muscle to a bone, complain of “pulled muscle,” severe cases cause inflammation, swelling, weakness and loss of function-surgery may be needed Management: PRICE (protect, rest, ice, compression, elevation), limitation ofactivity, physical therapy, NSAIDS, referral to ortho Dislocation- complete separation of 2 bones that form a joint Very painful and cause immobility, need immediate medical attentionReferral to orthopedics for possible surgery or reduction with application of cast or splint. 2. Signs and symptoms and management of spinal disorders (spondylosis, stenosis, etc.) Sandra Okonkwo Thank you Ashley L for Cervical Spondylosis- neck stiffness, mild aching discomfort with activity. Painand limited ROM occur with lateral rotation and lateral flexion of the neck toward the affected side. Weakness shoulder abduction- completing!!! C5. Bicep weakness-C6. Tricep weaknessC7.Myelopathy- leg weakness, gait disturbance, balanceproblems, difficulty performing fine motor tasks, loss of bowel and bladder. Treatment- cervical traction, PT, pain relievers. Surgery for Myelopathy. Low back pain-Tenderness and decreased range of motion. Positive straightleg test. Treatment-NSAIDS, muscle relaxants, opioids, surgical, self-care, spinal manipulation Stenosis-pseudoclaudication causing radicular pain in the calves, buttocks, andupper thighs of one or both legs. Symptoms progress from a proximal to distal direction. Walking or prolonged standing causes pain and weakness in buttocks and legs. Stooping over helps relieve pain. Positive Romberg. Reflexes diminished. Management- surgical decompression. NSAIDS, folic acid, vitamin b12. PT-flexing the spine.Bicycling. 3. Recognition and immediate management of cauda equina syndrome Danie Molly Immediate management of cauda equina syndrome. (P. 829) Cauda equina syndrome is a medical EMERGENCY and requires immediatedecompression. If Cauda equina is confirmed, surgical lumbar decompression is necessary tohalt neurological deterioration unless surgery is contraindicated for other medical reasons. *Rational on Davis Edge question: Low back pain accompanied by acute onsetof urinary retention or overflow incontinence, loss of anal sphincter tone or fecal incontinence, loss of sensation in the buttocks and perineum, and motorweakness in the lower extremities is a red flag for cauda equina syndrome or severe neurologic compromise 4. Maneuvers and expected findings with joint pain (knee, shoulder, wrist,etc.) Deanna Morrison Thank you Ashley L!! Neck pain-Spurling’s. Shoulder pain-Apley scratch test(reaching the scapula).Internal and external flexion. Internal and external abduction. Pain with abduction= early supraspinatus tendinitis and subacromial bursitis=early rotator cuff injuries. Wrist and hand-allen’s test= radial and ulnar arteries. Phalens test=median nerve compression. Tinel’s sign assess for compressionneuropathy. Finkelsteins test- de Quervains disease. Knee Pain= Mcmurray, apprehension sign, bulge sign, inspect/palpate to assess effusion. 5. Initial assessment of FOOSH injury in correlation to anatomical location of radial head bone Lisa Callahan FOOSH: Falling On an Out Stretched Hand. After falling on an outstretched hand patients present after trauma with pain and swelling in the distal forearm or wrist. Numbness may be present if the medial nerve is affected. The mechanism of injury will often provide important clues to the diagnosis. The examination begins with gentle palpation to locate the area of point tenderness and includes a thorough neurovascular assessment. A radiograph of the wrist (including an oblique view) may be necessary to rule out fracture.Common fractures are the Colles fracture of the distal radius and the navicular (scaphoid) fracture of the anatomical snuffbox. It is not unusual to have a navicular fracture missed on radiography, so an orthopedic referral should be provided when the presenting complaint is pain and trauma to the soft-tissue area of the anatomical snuffbox. 6. Assessment and management ofMyofascial pain Jenna Lara Thank you Ashley L Trigger points within a muscle. Common cause of nonarticular rheumatic pain.Injections a the trigger point with saline, an anesthetic, or corticosteroid, dry needling, muscle relaxant tizanidine, NSAIDS, or cyclooxygenases-2 inhibitors. Tricyclic antidepressants. 7. Health promotion activities toprevent sport related musculoskeletal injuries Melissa Schaf Protection may refer to preventing the injury from occurring or making it lesssevere by wearing protective gear, such as helmets, wrist pads, and kneepads.Maintain adequate hydration and proper diet while playing sports. Stretch before the activity. Stop when you are injured, do not “tough it out”. 8. Osteopenia Helena Longfellow Osteopenia: • Is the precursor to osteoporosis. Osteopenia is categorized by the levelof T-scores in relation to the results of a dual-energy x-ray absorptiometry scan or (DXA Scan), which measures the mineral content of bone. A T-score ranging from -1 to -2.5 would be classified as osteopenia. Pathophysiology: • It occurs secondary to uncoupling of osteoclastosteoblast activity, resulting in a quantitative decrease in bone mass. Peak bone mass is typicallyachieved by males and females just prior to, or early-on in the 3rd decade oflife. • Beyond age 30, bone resorption gradually becomes favored asdynamic bone remodeling continues into later decades of life. • Histologic specimens demonstrate markedly thinned trabeculae,decreased osteon size, and enlarged haversian and marrow spaces. Osteopenia Prevention: • Certain habits can accelerate the process such as:o Smoking • Not getting enough calcium and vitamin D • Drinking too much ETOH • Use of certain medications (i.e.: corticosteroids and anticonvulsants) • Not getting enough weight-bearing exercise (at least 30 mins on mostdays). If your feet tough the ground during an exercise, it’s probably weight bearing. Running and walking are weight bearing. Swimming and biking are not • Falls • Women are more likely to have low bone density than men, but it’s nolonger viewed as solely a women’s condition. • Approx. a third of white and Asian men over age 50 are affected. • Percentages for Hispanics (23%) and blacks (19%) are lower, but stillsizable. Current National Osteoporosis Foundation (NOF) recommends testing for: • Women 65 and older • Postmenopausal women younger than 65 who have one or more riskfactors, which include being thin • Postmenopausal women who have had a fracture • For men: testing is done more on a case-by-case basis. Osteopenia Treatment: Can be treated with exercise and nutrition or with medications. • If T-score is under -2, need to ensure you are doing regular weight-bearing exercise, and getting enough vitamin D and dietary calcium. • If T-score is closer to -2.5, a medication may be considered to keepbones strong. • Bisphosphonates are most commonly prescribed medication class fortreatment. Prolonged use has been linked with 2 major clinical side effects: osteonecrosis of the jaw (ONJ) and the atypical subtrochanteric femur fracture. • ONJ is rare and is associated with IV forms and not oral forms of themedication. Tx entails immediately stopping the offending agent. • Atypical femur fractures also are rare but have significant associated morbidity, and clinicians are cautioned against the chronic, uninterrupted bisphosphonate use beyond 3 to 5 years or in situations when pts report mildthigh discomfort while undergoing tx. • The core treatment options for osteopenic patients involve early education on how to achieve and maintain healthy bone mass levels and extensive education and counseling on the relevant social, environmental, andlifestyle risk factors that compromise bone health. General consensus favors pharmacologic treatment in a patient with spine orhip fractures in addition to a documented low BMD. Treatment recommendations vary for other nonvertebral fractures and include the following: • The National Osteoporosis Society (NOS) recommends starting treatment in all postmenopausal women with a history of any fragility fracture • The National Osteoporosis Foundation (NOF) recommends performingDXA scans on patients sustaining nonvertebral fragility fractures, and the decision to treat or not with pharmacotherapy is based on the patient’s t- score; patients considered to be osteopenic (t-score between -1 and - 2.5) arenot started on drugs. Pharmacotherapy agents work through either anti-resorptive or anabolic means. Bisphosphonates are the most commonly prescribed medication class.These drugs are divided into nonnitrogen and nitrogen-containing compounds. The latter are considered first-line therapy. The nitrogen- containing compounds inhibit farnesyl pyrophosphate synthase and ultimatelyinhibit osteoclast resorption and induce osteocyte apoptosis. Common agentsinclude: • Alendronate may reduce the rate of hip, spine, and wrist fractures by50% • Risedronate may reduce vertebral and nonvertebral fractures by 40%over three years • IV zoledronic acid reduces the rate of spine fractures by 70% and hipfractures by 40% over three years Other Medication Classes • Conjugated estrogen-progestin hormone replacement